Question 19

Compare and contrast viral, prokaryotic and eukaryotic genomes, and describe some mechanisms by which gene expression is regulated in each cell type. [Chapters 18 and 19]

                                                                         

The Genetics of Viruses (read pages 328-331)

 

Researchers discovered viruses by studying a plant disease.

 

A virus is a genome enclosed in a protective coat.

 

Viruses can reproduce only within a host cell.

 

Phages reproduce using lytic or lysogeneic cycles.

 

Animal viruses are diverse in their modes of infection and replication.

 

Plant viruses are serious agricultural pests.

 

Viroids and prions are infectious agents even simpler than viruses.

 

Viruses may have evolved from other mobile genetic elements.

 

The Genetics of Bacteria (read pages 340-351)

 

The short generation span of bacteria helps them adapt to changing environments.

 

Genetic recombination produces new bacterial strains.

 

The control of gene expression enables individual bacteria to adjust their metabolism to environmental change.

 

Genome Organization at the DNA Level (pages 357-362)

 

Repetitive DNA and other noncoding sequences account for much of a eukaryotic genome.

 

Gene families have evolved by duplication of ancestral genes.

 

Gene amplification, loss or rearrangement can alter a cell’s genome during an organism’s lifetime.

 

The Control of Gene Expression (read pages 362-368)

 

Each cell of a multicelluar eukaryote expresses only a small fraction of its genes.

 

The control of gene expression can occur at any step in the pathway from gene to functional protein.

 

Chromatin modifications affect the availability of genes for transcription.

 

Transcription initiation is controlled by proteins that interact with DNA and with each other.

 

Post-transcriptional mechanisms play supporting roles in the control of gene expression.

 

The Molecular Biology of Cancer (read pages 369 to 372)

                                                    

Cancer results from genetic changes that affect the cell cycle.

 

Onocogene proteins and faulty tumor-suppressor proteins interfere with normal signaling pathways.

 

Multiple mutations underlie the development of cancer.

 

 

 

John C. Murphy

           Science

Phone:  (815) 436-3200, ext. 2120

 

jmurphy@learningcommunity202.org

 


 

Plainfield High School Home Page

Link to District 202 Home Page

Science Home Page

© 2001 Plainfield Community Consolidated School District 202.